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Multiple inflammatory cytokines, including TNF-, prostaglandin E2 (PGE2), IL-1, IL-6, and IL-1, have been shown to modulate GPX4 expression and activity [165]

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After 4 days of activation with anti-IgM, CD40 ligand, and IL-4, we found that blocking of mitochondrial metabolism with mitochondrial inhibitors or blocking GSH synthesis by BSO had lowered antibody production of FoB in a dose-dependent manner (Fig

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