Discussion In this study, we showed that cigarette smoke exposure, even without the presence of tobacco, promoted lung inflammatory injury, as indicated by the presence of inflammatory cells in BALF and oxidative stress (measured by TBARS levels, a reduced GSH/GSSG ratio, and increased activity of antioxidant enzymes SOD and CAT)
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It may also be due to variations in how the question to assess flavored cigarette use was asked in the baseline and follow-up questionnaires, as described above
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