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While glucagon does raise blood sugar by stimulating the liver to release glucose, the GLP-1 and GIP components counterbalance this, resulting in net improvements in glucose control

Due to lack of stratified data by time points across studies, we were unable to perform a subgroup analysis based on duration of treatment, which limited our ability to evaluate time-dependent risk trends of gastrointestinal adverse events

We further observed that pretreatment with C1-27 inhibited the transcriptional induction of pro-IL-1 in response to inflammatory stimulus such as the bacterial endotoxin, lipopolysachcharide

NAC acts as an antioxidant by increasing GSH levels and by directly interacting with free radicals