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Benefits of knocking down YTHDF1 on GCs are reversed by SLC7A5 overexpression The protective effect of YTHDF1 on GCs and its molecular mechanism were systematically evaluated by knocking down YTHDF1 and overexpressing SLC7A5

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10.1007/5584_2019_381 Summary Keywords osteoarthritis, chondrocyte ferroptosis, synovial inflammation, regenerative therapy, meniscus degeneration Citation Qiu L, Alhaskawi A and Moqbel SAA (2026) Osteoarthritis: multitissue pathology, molecular mechanisms, clinical management, and emerging precision and regenerative therapies

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