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Reduced Risk of Nutritional Gaps Rapid weight loss can sometimes lead to nutrient deficiencies, especially when meals become smaller or less varied

Supports transition from preclinical to clinical IBD research Favorable safety profile across all preclinical toxicology studies Effective in TNBS-colitis and cysteamine-duodenal ulcer models

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Phosphorylation of IRS-1 or IRS-2 activates phosphoinositol 3-kinase (PI3K)-PDK1-AKT signaling pathway, while phosphorylation of Shc leads to activation of RAS, RAF and extracellular signal-regulated kinase (ERK)/mitogen-activated protein kinase (MAPK) signaling pathway