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Retreatment of patients who had virologic failure to G/P therapy may require a regimen with more than two mechanisms of action, such as G/P plus SOF, which is a DAA combination currently being evaluated in an ongoing clinical study (registered clinical trial: NCT02939989)

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in the economically developed world, however, the widespread availability and the absence of overt deficiency states give little cause for most individuals and their caregivers to pay attention to these potentially pathophysiologic vulnerabilities [4]

The enzyme uses glutathione as the ultimateelectron donor to regenerate the reduced form of the selenocysteine