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10.1016/j.jep.2024.118354 170 YinZ.TanR.YuanT.ChenS.QuanY.HaoQ.et al (2021)

It also suggests that once mitochondrial injury has been initiated, its further progression may become increasingly independent of MCU itself owing to self-sustaining feed-forward mechanisms such as mPTP opening, oxidative stress, defective Ca 2+ extrusion, and inflammatory signaling